ces-03-181-g002
p53- and Drug-Induced Apoptotic Responses Mediated by BH3-Only Proteins Puma and Noxa | Science
Phylogenetically Distant Viruses Use the Same BH3-Only Protein Puma to Trigger Bax/Bak-Dependent Apoptosis of Infected Mouse and Human Cells | PLOS ONE
Life | Free Full-Text | BH3-Only Proteins Noxa and Puma Are Key Regulators of Induced Apoptosis
Phylogenetically Distant Viruses Use the Same BH3-Only Protein Puma to Trigger Bax/Bak-Dependent Apoptosis of Infected Mouse and Human Cells | PLOS ONE
A review of the role of Puma, Noxa and Bim in the tumorigenesis, therapy and drug resistance of chronic lymphocytic leukemia | Cancer Gene Therapy
BAX and BAK become killers without a BH3 trigger | Cell Research
BID, BIM, and PUMA Are Essential for Activation of the BAX- and BAK-Dependent Cell Death Program | Science
Phylogenetically Distant Viruses Use the Same BH3-Only Protein Puma to Trigger Bax/Bak-Dependent Apoptosis of Infected Mouse and Human Cells | PLOS ONE
Application of cryo-electron microscopy for investigation of Bax-induced pores in apoptosis
BH3-dependent and independent activation of BAX and BAK in mitochondrial apoptosis - ScienceDirect
Bax/Bak activation in the absence of Bid, Bim, Puma, and p53 | Cell Death & Disease
Bax/Bak activation in the absence of Bid, Bim, Puma, and p53 | Cell Death & Disease
BH3-dependent and independent activation of BAX and BAK in mitochondrial apoptosis - ScienceDirect
Interplay of Bcl-2 Proteins Decides the Life or Death Fate
Models for how the BH3-only proteins activate Bax and Bak. (A) In the... | Download Scientific Diagram
Bax/Bak activation in the absence of Bid, Bim, Puma, and p53 | Cell Death & Disease
Both p53–PUMA/NOXA–Bax-mitochondrion and p53–p21cip1 pathways are involved in the CDglyTK-mediated tumor cell suppression - ScienceDirect
Spontaneous oligomerization of BAK/BAX is suppressed by hetero-dimerization with MCL-1 | bioRxiv
BID, BIM, and PUMA Are Essential for Activation of the BAX- and BAK-Dependent Cell Death Program | Science
BAK/BAX-Mediated Apoptosis Is a Myc-Induced Roadblock to Reprogramming
Investigation of Bax-VDAC Interactions and Their Role in Apoptosis
